Page 1 of 1: Ganong Corner-Discuss all your doubts from Ganong!

super7 · Credits: 2113 My Scrapbook

Keep posting all ur doubts and important topics from Ganong[or other Physiology queries] and may be we can solve them together...........

Seniors preparing for their PG can help us in telling imp. topics to be stressed upon in Physio.

I'll be posting some soon.....

super7 · Credits: 2113 My Scrapbook

1.{Chap1 Pg24[Ganong,22nd edition]}:m-RNA is stabilised by poly-A tail.Where exactly is this poly-A present.In the diag.,they have shown it on the DNA strand.But still it is also prsent at the untranslated segment at the 3' end.How is this possible?

2.{Chap1 Pg41]}:Ca sparks :Some of the changes it produces can outlast the rise in intracellular Ca conc. because of the way it binds to Ca-binding proteins.........what exactly does he mean?

3.This one is general:Why does our R.B.C count decrease in the morning and increase in the evenings??

jeanie · Credits: 529 My Scrapbook

how does hyperkalemia l/t decreased rmp and stabilise the membrane.+what does decreased rmp mean-towards +???

clearcut · Credits: 2084 My Scrapbook

Hi everyone.
I`ve a doubt .
During ventricular systole , the pressure wave generated in aorta reaches the radial artery in 0.1 sec after the point of maximum pressure in aorta , in normal adult which we perceive as radial pulsation . This pressure wave has got nothing to do with blood flow to the radial artery(which is quite slower compared to pressure wave). Also during systole the aorta stretches n this results in its recoiling during diastole.
My doubt is- as aorta recoils during diastole, its gonna send a pressure wave again ( by the same principle with which pressure wave during systole is generated), So why don`t we feel second pulse (of diastole) within the same one cardiac cycle ?

With regards.

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