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shankardada
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FISHMOUTH VERTEBRA
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07.14.04 (4 years ago)
#1
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where is it seen?In salgu its given iN HOMOCYSTINURIA
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RxPG_Team
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07.14.04 (4 years ago)
#2
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The “fish-mouth” vertebrae are caused by vaso-occlusion in Sickle cell anaemia. Fish-mouthed vertebrae are formed because short, perforating branches of the periosteal vessels that supply the periphery are not as
prone to vaso-occlusion as the long branches of the vertebral nutrient arteries that supply the center of the end plate. Vaso-occlusion are more likely to occur the longer erythrocytes spend deoxygenating in the cramped, acidic environment of the capillaries. Therefore, the edges of
the vertebrae extend normally because their vascular supply is less frequently compromised; whereas, the centers of the vertebrae are depressed and introverted.
It is also seen in Gaucher's Disease
See Chapman's radiology for a reference.
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RxPG_Team
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07.14.04 (4 years ago)
#3
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fish-mouth mitral stenosis: Extreme mitral stenosis.
fish-mouth meatus: A red and swollen condition of the orifice of the urethra (urinary meatus) in gonorrhoea.
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RxPG_Team
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07.14.04 (4 years ago)
#4
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| RxPG Team wrote: |
The “fish-mouth” vertebrae are caused by vaso-occlusion in Sickle cell anaemia. Fish-mouthed vertebrae are formed because short, perforating branches of the periosteal vessels that supply the periphery are not as
prone to vaso-occlusion as the long branches of the vertebral nutrient arteries that supply the center of the end plate. Vaso-occlusion are more likely to occur the longer erythrocytes spend deoxygenating in the cramped, acidic environment of the capillaries. Therefore, the edges of
the vertebrae extend normally because their vascular supply is less frequently compromised; whereas, the centers of the vertebrae are depressed and introverted.
It is also seen in Gaucher's Disease
See Chapman's radiology for a reference. |
More on sickle cell disease bone changes:
Repeated cycles of bone infarction following sickling-induced anoxia, with or without secondary osteomyelitis, and associated marrow hyperplasia
induced by chronic hemolysis all contribute to chronic bone Pathology
in SCD. The “vanishing of vertebrae,” spondylitis, pseudo-Schmorl’s nodes, narrowed intervertebral disc spaces, and fish-mouth deformities of vertebral bodies seen in SCD can be explained on the basis of vaso-occlusive infarction, secondary infection, compressive forces along the spine, and reactive bone processes. Although it is called the “vanishing vertebra,” this phenomenon is only an apparent radiologic disappearance,
as the radiolucent osteoid of the “vanished” vertebra will necessarily still be present in the spine.
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drmalik
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hi buddy have u some picture slides etc of fishmouth vetebra
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07.18.04 (4 years ago)
#5
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hi buddy have u some picture slides etc of fishmouth vetebra
   
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nadu
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07.18.04 (4 years ago)
#6
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Originally coined for the changes of senile osteoporosis, biconcave deformities of the vertebral bodies ("fish vertebrae") are characteristic of disorders in which there is diffuse weakening of the bone. The name is derived from the actual appearance of a fish vertebrae which normally has depressions in the superior and inferior surfaces of each vertebral body. This sign is typically used for osteopenia.
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nadu
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07.18.04 (4 years ago)
#7
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Central depression of multiple vertebral end-plates: a "pathognomonic" sign of sickle hemoglobinopathy in Gaucher's disease.
Lateral radiographs of the spine reveals central endplate depression with sparing of the anterior and posterior margins of the endplate. Several current names have been ascribed to these same changes seen in patients with hemoglobinopathy, such as Lincoln Log Sign, or H-shaped vertebra. Although not pathognomonic, these terms are typically reserved for a patient with Sickle-Cell disease.
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sumer sethi
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11.23.04 (3 years ago)
#8
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nice pics nadu!!! that was good one!!
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Nagu
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02.02.05 (3 years ago)
#9
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Dear Dr. Sethi and other colleagues,
16th edition Salgunan
. Could you give me the correct answers.
1. 1/2 life of technitium
a. 2hrs
b. 6 hrs
c. 12 hrs
d. 24 hrs
2. Lymphangitis carcinomatosa is a typical feature of
a. Ca. thyroid
b. Ca Bronchus
c. Ca pancreas
d. Hepatoma
e. Hypernephroma
3. Terminal phalangeal sclerosis is associted with
a. Rheumatoid arthritis
b. TB
c. Sarcoidosis
d. Pleural Fibroma
e. Chronic active hepatitis.
4. Hydrocephalus in children, first seen is -
a. Sutural diastasis
b. Posterior clinoid erosion
c. Large head
d. Thinned out vault
(also tell me what would be first seen in adults in Hydrocephalus)
5. Investigation of choice in Ac. Renal failure with anuria. USG is normal.
a. Renal angiography
b. DTPA
c. IVP
d. Retrograde pyelography
(this question with same choice was repeated in the same book but the answer was different (Q.305 and Q 279)
Please help
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DRATKINS
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03.06.05 (3 years ago)
#10
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Q1- t1/2 OF Tc is 6 hrs
Q2-LYMPHANGITIS CARCINOMATOSA IS SEEN IN bronchial & pancreatic Ca both
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