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docsatya76
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CMC Vellore
2008 questions gen paper
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01.05.08 (6 months ago)
#1
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1. sago spleen
a. amyloid deposition spleen sinusoids
b. '' '' '' follicles
c. tb spleen
d.
2. mc thyroid ca
a. papillary
b. medullary
c. anaplastic
3. ground glass appearance lungs lower lobe
a. asbstosis
b. silicosis
c. bagossis
4. pheochromocytoma monotherapy c/i
a. proprenolol
b.
5.
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rohkav12
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01.05.08 (6 months ago)
#2
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cystic hygroma is seen in....
1.turner
2.downs
3.klienfelters
4.edward
5.patau
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rohkav12
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01.05.08 (6 months ago)
#3
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hemolytic anemia is seen in
1.malaria
2.fanconi anemia
dilated pupil with absent direct n present consensual reflex is seen in...
1aryll robertson
2.marcuss gunn
3.3 nerve palsy....
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me2
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01.05.08 (6 months ago)
#4
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1.differential cyanosis is seen in ? 2.flexors of shoulder joint?
3.abductors of hip?4.waddling gait is seen in?
5.weils disease is caused by? 6.m.c trisomy?
7.mean distribution width is increased in which anemia?
8.broncholitis obliterans -main mode of treatment?
9.m.c cause of meningitis in newborn with hiv a;cryptococcal meningitis b;acute pyogenic meningitis?
10.plantar ulcer in leprosy causes a,septic arthritis b;teno vaginitis c,osteomyelitis?d...
11.which of the following causes c.o.p.d...a,amyloidosis b,alfa1 anti trpsin def c,sarcoidosis d,
12.iron deficiency anemia causes a,copper def b,thallesmia c,Heriditary spherocytosis?
13.lactose fermenting non motile bacteria.a,e.coli b,proteus c,klebsilla d,pseudomonas?
14,blood stained floor is sterilised by?
15,acute abdomen investigation of choice?
16,ocular manifestation of bechet syndrome/anky.spondylitis?]
17.m.c benign intra ocular tumour in adults a,capillary haemangioma b.cavernous haemangioma?
18.dysentry caused bya,E.COLI B,E.HISTOLYTICA C,NAEGLERIA
19,SCHIZO AFFECTIVE DISORDER-no mood symptoms
20,m.c cranial complication of o.media a,sub dural abscess b,extra dural abscess c,meningitis d,..?
21.clinical feature of o.media?
22.bleeding time is prolonged in a.F-8 Def b.vWB DISEASE c,...
23.cervical cancer stage 2b is managed by?
24,eye is most prone to hyperglycemic effect because of deficiency of a.aldose reductase b,g6pd df c,
25.lithium side effects?
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me2
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01.05.08 (6 months ago)
#5
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26,m.c cardiac anomaly in turners syndrome? 27,B-HCG level -trans abominal usg in pregancy?
27,reliable criterias -to diagnose fetal growth in 1st trimester a,CRL B,CRL+bpd c,FL...
28, 28 YEAR TERM PREGANCY DIAGNOSED TO HAVE NUCHAL CORD ,FETUS ALIVE.HOW TO MANAGE?
29,CRITERIA TO BE FULFILLED IN FORCEPS DELIVERY-RELATED TO STATION?
30,CHORIO CARCINOMA USALLY FOLLOWS A,TERM PREGANCY B.ABORTION C,H.MOLE D,...
31.SULFONAMIDE SIDE EFFECT A,ERYTHEMA INDURATUM B.E.NODOSUM C,E.MARGINATUM D,,
32,LIPID NECROSIS IS SEEN IN A,A.PANCREATITIS..B,
33,APOPTOSIS HISTOLOGICAL FEATURES ?
34,NODE OF CLOQUET IS SEEN IN A,FEMORAL CANAL B,INGUINAL CANAL ..?
35,DRUG C.I IN LIVER INJURY A,CYCLOPHOSMAMIDE B,RIFAMPICIN C,..
36,PROSTATE CARCINOMA MEDICAL MANAGEMENT?RELATED TO DRUG USED.?
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potya
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medicine
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01.05.08 (6 months ago)
#6
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28 yr female with hiv on trtmnt with nevirapin,stavdine,lamivudine since 9mnths.cd count550/litre,hiv rna<50(1 month back)since 3 wks c/o wt loss anorexia, jaundice.o/e liver span10 cms in midclavicular line dignosis?
1 drug inducd hypersensi
2immunorecostitutive syndrome
3candidiasis
4drug toxicity
plz reply urgent
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nadu
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01.05.08 (6 months ago)
#7
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keep it up
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doctorbychance
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regarding the hiv question
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01.05.08 (6 months ago)
#8
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i personally think it is drug toxicity . well if it was drug induced hypersensitivity , then it wud not have manifest after so many months . and regarding the other options , i think candidiasis is not related to hepatomegaly . anyway i wud like to know the correct answer too, coz i marked it as drug toxicity . any masters out there we are waiting . anyway this is wat i think potya.
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inayath
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01.05.08 (6 months ago)
#9
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The term "immune reconstitution inflammatory syndrome" (IRIS) describes a collection of inflammatory disorders associated with paradoxical worsening of preexisting infectious processes following the initiation of highly active antiretroviral therapy (HAART) in HIV-infected individuals
Preexisting infections in individuals with IRIS may have been previously diagnosed and treated or they may be subclinical and later unmasked by the host's regained capacity to mount an inflammatory response.
Symptoms and laboratory abnormalities suggesting worsening hepatitis may occur in patients coinfected with HCV or HBV following HAART
Affected patients typically have elevated serum liver enzymes accompanied by fevers, night sweats, anorexia, nausea, fatigue, tender hepatomegaly, and jaundice
IRIS associated with hepatitis viruses B and C usually occurs within two to eight weeks of initiation of HAART but onset may be delayed for up to nine months
ABOUT Drug Toxicity :
Nucloside Reverse Transcripters : Stavudine , Lamivudine.
have been associated with the disorder of lactic acidosis/severe hepatomegaly with steatosis.
In the Question Severe Hepatomegaly is mentioned.
At 28 years female.........in the question no preexisting infection is mentioned like hepatitis C /B...
Duration of treatment too is for 9 months.........
The above 3 points favour drug toxicity rather.......Immune reconstitution inflammatory syndrome ( IRIS)..
but......
symptoms since 3 weeks with HIV load < 50 again points towards .......paradox of IRIS.
Was there anything more in the question description ........???? and if the duration of the treatment is 9 months.........it will be more in favour of Drug toxicity.
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coolniks
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hi
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01.05.08 (6 months ago)
#10
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well que to sulfonamide...
sulfonamide casues the following skin reaction
erythem marginatum.
erythema nodosum
eythma multiforme.
4th..??
ans is
ERYTHEMA MULTI FORME...
ref..http://www.medscape.com/viewarticle/434455[/u]
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